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For Research Use Only — Not for Human Consumption.
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Researched Benefits:
MOTS-C 10MG is a research peptide referenced in scientific literature as a mitochondrial-derived peptide, part of a group of bioactive peptides and mitochondrial derived peptides, and is classified as a mitochondrial encoded peptide. MOTS-c is studied as a metabolic regulator with broad physiological functions relevant to laboratory research, including its ability to enhance glucose metabolism, support cell function, and promote cell survival under metabolic stress.
This MOTS-C 10MG product page provides educational, research-only information focused on compound classification, laboratory context, and non-clinical investigation.
MOTS-C 10MG is supplied by NuRev Peptides strictly for laboratory research and testing purposes. All information presented here is neutral, factual, and intended to support scientific discussion rather than clinical, therapeutic, or diagnostic application. This compound is not intended for human or animal use.
MOTS-C 10MG is discussed in research environments as a peptide encoded by mitochondrial DNA, derived from a mitochondrial ORF, and classified as a mitochondrial encoded peptide. Research explores the molecular mechanisms of MOTS-c, including its ability to regulate nuclear gene expression through nuclear translocation and interaction with transcription factors and nuclear genes. MOTS-c expression and its effects on muscle cells, especially mouse skeletal muscle, are common research topics. Experimental models often involve mice fed a normal diet or high fat diet to study diet induced obesity and metabolic dysfunction. Systemic MOTS-c treatment and MOTS-c treatment are studied for their metabolic effects, including reducing insulin resistance, improving insulin secretion, and addressing obesity and insulin resistance. Research also measures plasma MOTS-c levels, circulating MOTS-c levels, and serum MOTS-c levels as biomarkers. Cellular uptake of MOTS-c is investigated to understand its biological activity. MOTS-c’s role in glucose homeostasis, fatty acids metabolism, and cell metab is also a focus. Additional research explores MOTS-c’s effects on reactive oxygen species, mitochondrial dysfunction, aging cell, adaptive responses, muscle homeostasis, and cardiovascular disease. Glycosylated hemoglobin is used as a research endpoint. MOTS-c is studied as an endocrine factor and metabolic regulator, with research on MOTS-c expression and the peptide MOTS-c mots. Investigation of immune response, inflammatory markers, and proinflammatory cytokines in relation to MOTS-c is ongoing. Human studies are conducted to assess the physiological relevance of MOTS-c.
Research discussions involving MOTS-C 10MG are limited to laboratory-based investigation and experimental research models. References focus on biochemical pathways, signaling interactions, and molecular behavior rather than clinical outcomes or approved applications. NuRev Peptides does not make claims regarding efficacy, safety, or results related to this compound.
All information on this page is provided strictly for educational and research discussion purposes. This compound is not intended for diagnostic, therapeutic, medical, or veterinary use under any circumstances.
NuRev Peptides maintains a research-only policy and emphasizes transparency, documentation, and third-party laboratory verification where applicable.
Endogenous MOTS-c is the naturally occurring form of the peptide mots c, produced within the body as a mitochondrial-derived peptide. This bioactive molecule is encoded by the mitochondrial genome and is recognized for its essential role in supporting metabolic homeostasis and regulating mitochondrial function. Research in laboratory settings has demonstrated that endogenous mots c levels are closely linked to the body’s ability to adapt to metabolic stress, with the derived peptide mots c contributing to the maintenance of cellular energy balance and overall metabolic health.
Studies indicate that as organisms age, endogenous mots c levels tend to decline, which may be associated with age dependent physical decline and increased susceptibility to metabolic stress. This reduction in mots c peptide availability can impact the efficiency of mitochondrial function and disrupt the delicate balance required for optimal metabolic homeostasis. Ongoing research continues to explore how fluctuations in endogenous mots c influence physiological processes and the potential implications for age-related metabolic changes.
Additional scientific context related to compounds can be found through publicly available research databases such as PubChem.
For further information on purity please visit Certificates of Analysis.
To learn more about our research disclaimers please visit our page Research-only disclaimer.
Research peptides are amino-acid chains used in labs to study how the body works. They are for research use only—not for human or animal consumption.
No prescription is required. By purchasing, you confirm products will be used strictly for research purposes only and in compliance with applicable laws and regulations.
For long-term storage, lyophilized peptides should be kept in the freezer. After reconstitution, store peptides in the refrigerator and use promptly for research.
We do not provide usage or dosing instructions. Products are for research/testing only, and no medical or experimental guidance is offered.
Most orders ship within 1–2 business days. You’ll receive a tracking number when your order ships. Transit times depend on the carrier and destination; weather or carrier issues may cause delays.
At this time, we ship within the United States. If international shipping becomes available, we’ll announce it on our website.
We can modify or cancel an order only if it hasn’t been processed or shipped. Email [email protected] with your order number as soon as possible.
Due to the nature of our products, all sales are final. We’ll replace or refund if an item arrives damaged, defective, or incorrect (contact us within 7 days of delivery with photos and your order number). See our full refund policy here.
